Sickle Cell Anemia is a disorder caused by at least one of the hemoglobin sub-units, beta-globin being replaced with hemoglobin S. Sickle cell anemia is a co-dominance gene. If both parents have heterozygous normal genes a child is capable of having Sickle cell anemia.
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Sickle Cell Anemia Symptoms

  • Severe pain in the chest, joints, bones, and abdomen
  • Difficulty breathing
  • Blockage of flow in the spleen or liver
  • Severe infections
  • Fatigue
  • Fever
  • Paleness
  • Ulcers on the lower legs

Treatments and preventions of death may include:
  • Folic acid supplements
  • Blood transfusions
  • Pain medication
  • Fluids
  • Hydroxyurea(Hydrea)
  • Antibiotics
  • Drug rehabilitation
  • Kidney dialysis
  • Surgeries if neccesary

Tests performed to diagnose or monitor patients with sickle cell anemia:
  • Complete blood count
  • Hemoglobin electrophoresis
  • Bilirubin
  • CTI or MRI scan
  • Blood oxygen
  • Pap smear
  • Serum creatinine
  • Serum hemoglobim
  • Serum pottassium
  • Urinary casts
  • White blood cell count
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  • Reduced life expectancy There is no prevention for Sickle Cell Anemia other than parents can find out if their child will have Sickle Cell Anemia by getting a genetic screening and can choose whether or not to have the child.
    • 1973 the average life expectancy was 14
    • Now people with sickle cell anemia live to be in their 40s and 50s sometimes 60s

  • Sickle cells have a lower life span and only live 10-20 days
    • The bone marrow can not replenish the dying sickle cells with the amount dying
    • People diagnosed with sickle cell have a lower blood count


  • Contributors. " Sickle Cell Anemia- Health -" Inspiration, Spirituality, Faith, Religion. - N.p., n.d. Web. 27 Feb. 2012. <>.
  • Newton, David E.. "Sickle Cell Anemia." Sick! diseases and disorders, injuries and infections. Detroit: U·X·L, 2000. 676. Print.